Tinnitus
• Incidence 10-20% of population
• Pulsatile vs non-pulsatile
• Hearing loss (Most common by far)
Common causes
• Unknown 23%
• Arterial stenoses 18%
• Intracranial hypertension 16%
• Venous anatomical variant 12%
• Other 8%
• Dural AVF 7%
• Vessel rich tumour (AV connection) 6%
• Arterial anatomical variants 4%
• Capillary hyperaemia 3%
• Direct AVF 2%
• Aneurysm 1%
Full list
• Tumour
o Paraganglioma (most common)
- Glomus typanicum or glomus jugulare (auricular branch of nerve)
o Meningioma
o Endolymphatic sac tumour
o Other skull base tumours
• Vascular – Arterial
o Aberrant ICA
o Persistence of stapedial artery
o ICA dehiscence
o Persistent trigeminal artery
o Arterial stenosis or dissection
o Dural AVF
o Aneurysm (very rare)
o Vascular loop in IAM (non a proven link)
• Vascular – venous
o Transverse sinus stenosis
- Intrinsic (Arachnoid granulation/thrombosis)
- Extrinsic (IIH)
o Sigmoid sinus wall anomalies (dehiscence or diverticulum) - ?very common cause
- Outpouching of sigmoid sinus into mastoid
o Emissary vein enlargement
o Jugular bulb abnormalities (most probably incidental)
- High riding jugular bulb (above level of IAM)
- Dehiscence
• Bone abnormalities/dysplasia
o Otospongiosis
o Paget;s
o Facial nerve venous malformation
o Superior semicircular canal dehiscence
• Bruit on auscultation - ?arterial cause
• Cessation of tinnitus with compression of IJV – venous cause
• Depends on cause
• Low yield in standard tinnitus
• Higher yield if unilateral hearing loss or pulsatile
• Pulsatile:
o MRA/MRV
o CT skull base

Dtsch Arztebl
Int 2013; 110(26): 451-8.
DOI: 10.3238/arztebl.2013.0451